Más allá de la actividad antioxidante, hacia la modulación redox: N-Acetilcisteína (NAC) en la endometriosis y los leiomiomas uterinos

anti-inflammatory properties that may target key mechanisms underlying both conditions. NAC functions as both a direct reactive oxygen species scavenger and regulator of intracellular redox homeostasis through replenishment of glutathione stores. These findings suggest that NAC may represent a potential adjunctive therapy warranting further investigation and may influence disease-associated biological pathways. Future clinical and translational studies are needed to define its efficacy, optimal dosing, and role in redox-based management of endometriosis and uterine leiomyomas.